用氨基甲酸乙酯（１．０ｇ／ｋｇ体重）腹腔麻醉ＳＤ大鼠，以膈肌放电和气管内压作为呼吸的指标。脑内注入微量１％ｐｒｏｃａｉｎｅ，双侧对称地阻滞闩前１．８－２．０ｍｍ，中线旁开１．８－２．０ｍｍ，背侧表面下２．８－３．３ｍｍ区域后，全部动物都出现了可逆性呼吸停止。用同样方法阻滞孤束核区，疑核区和延髓头端区对节律性呼吸无明显影响。经组织学检查，阻滞引起呼吸停止的区域为面神经后核及其内侧的区域，其范围为０．８ｍｍ×０．８ｍｍ×０．８ｍｍ。 SD rats were intraperitoneally anesthetized with urethane (1.0g / kg body weight), and diaphragmatic discharge and endotracheal pressure were used as indices of respiration. Intracerebral microinjection of 1% procaine bilaterally symmetrically block the front of the latch 1.8-2.0mm, 1.8-2.0mm next to the midline, 2.8-3.3mm area below the dorsal surface, all Animals have shown reversible respiratory arrest. Using the same method to block the nucleus tractus solitarius, the nucleus accumbens and the rostral region had no significant effect on rhythmic respiration. Histologically, the block of the area causing respiratory arrest was the posterior facial nucleus and its medial area, which ranged from 0.8 mm × 0.8 mm × 0.8 mm.