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有些慢性肾病患者,血钾很高而氮血症不明显,不好用肾功能减退来解释。这些病人往往有高氯性酸中毒和高血压。实验研究显示多数患者血浆肾素活性降低且醛固酮生成减少。多数作者认为是由于肾小球旁器损坏致肾素分泌减少,继发性地引起醛固酮分泌减少。本文作者报导4例慢性肾病患者,他们的肾脏病变的类型还不能肯定,但其中2例有痛风,2例有糖尿病并发的肾血管病变和慢性间质性肾炎。静脉肾盂造影2例肾脏明显缩小,1例肾脏大小正常,1例肾
Some patients with chronic kidney disease, high blood potassium and azotemia is not obvious, not to explain the use of renal dysfunction. These patients often have high chloride acidosis and high blood pressure. Experimental studies have shown that most patients have decreased plasma renin activity and decreased aldosterone production. Most authors believe that is due to damage to the glomerular perirenal secretion caused by reduced renin secretion, resulting in secondary reduction of aldosterone secretion. The authors report four patients with chronic kidney disease, their type of renal disease is still not sure, but two cases of gout, two cases of diabetic patients with renal vascular disease and chronic interstitial nephritis. Intravenous pyelography in 2 cases of renal significantly reduced, 1 case of normal kidney size, 1 case of kidney