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“通脉灵”在治疗硬皮病,烧伤颜痕等结缔组织性疾患中,有抑制成纤维细胞亢进的胶元合成作用,对实验性大鼠琼脂肉芽肿结缔组织也表现出同样作用,并使胶元量降低,但对不同溶解度胶元的比例却无影响。这表明“通脉灵”可能作用于胶元合成细胞内过程,这可能是它抗炎的结果。因在动物实验中许多抗炎药物抑制炎症,其结果均可导致肢无量的降低。 1971年Vane等证明非甾体抗炎药的抗炎作用是通过抑制前列腺素(Prostaglan-dlins,简称PGS)的生物合成。激素类抗炎药的作用机理也与PGS有关。而“通脉灵”
“Tong Mai Ling” in the treatment of scleroderma, burns and other signs of connective tissue lesions, there are inhibition of fibroblast hyperplasia synthesis of the role of the role of the rat rat agar granulomatous connective tissue also showed the same effect, and Reduce the amount of glue, but it has no effect on the proportion of glue with different solubility. This shows that “Tong Mai Ling” may act on the intracellular process of collagen synthesis, which may be the result of its anti-inflammatory. Because many anti-inflammatory drugs inhibit inflammation in animal experiments, the results can lead to an unpredictable loss of limbs. In 1971, Vane et al. demonstrated that the anti-inflammatory effect of non-steroidal anti-inflammatory drugs was through the inhibition of prostaglandin-like (PGS) biosynthesis. The mechanism of action of hormonal anti-inflammatory drugs is also related to PGS. And “Tongmai Ling”