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1984年5月我所收治了一例急性一氧化碳中毒的迟发性脑病病人。患者系吉林省夹皮沟金矿工程师,男性,44岁,于1984年2月29日夜,因火坑泄漏吸入一氧化碳12小时,当其家属晚班下班回家始发现,遂送当地医院抢救,昏迷达22小时。经抢救苏醒后,记忆力和思维均正常,七天后生活自理,且能阅读书报,商谈工作,住院14天,停药出院,自己步行回家。病假期间在家看书,还能设计制图。20天后突然出现无故发笑、反应迟纯、行为单一离奇,进而答非所问,故再次入院。虽经有关治疗,病情仍继续发展,定向力丧失、不识熟人、少语、继至失语、大小便失禁、意识丧失、肌张力增高呈木僵状态,住院47天,因病情加重而转我所治疗。经检查脑电图示弥漫性慢活动,脑波失律。CT示额叶脑白质广泛软化,
In 1984 May I admitted to a case of acute carbon monoxide poisoning in patients with delayed encephalopathy. The patient was a male engineer at Jiapigou Gold Mine, male, 44 years old. On the night of February 29, 1984, 12 hours after inhalation of carbon monoxide due to a fire pit leak, his family members came home from work after night shift and were sent to a local hospital for rescue and coma Up to 22 hours. After rescuing wake up, memory and thinking are normal, seven days after taking care of themselves, and can read newspapers, discuss work, hospitalized 14 days, withdrawal from hospital, walk home. Reading at home during sick leave, but also design drawings. 20 days after the sudden appearance of no reason to laugh, reaction to pure, unique bizarre behavior, and then give an irrelevant answer, so re-admission. Although the treatment, the condition continues to develop, loss of orientation, do not know acquaintances, few words, following aphasia, incontinence, loss of consciousness, increased muscle tension was stupor, hospitalized 47 days, due to aggravating and turn me Healed. After checking EEG showed diffuse slow activity, brain wave loss of law. CT showed frontal lobe white matter softened extensively,