十二指肠溃疡(Du)和胃溃疡(Gu)的发病机制具有异质性,除了Shay和Sun有关粘膜防御因子和攻击因子间失平衡的理论,还有上皮促生长因子、尿抑胃肽等因素在发病中有意义.本文拟就与消化性溃疡发病有关的另外几个方面作一简要回顾.I型单纯疱疹病毒(I-HSV)感染与消化性溃疡Warren等从迷走神经节中分离到I-HSV.潜伏的I-HSV经激活后可沿迷走神经到达消化道,引起疱疹性食管炎等消化道疱疹病变.此类病变在免疫功能低下的基础上发生,一般 The pathogenesis of duodenal ulcer (Du) and gastric ulcer (Gu) is heterogeneous except Shay and Sun’s theory of imbalance between mucosal defensive factor and aggressive factor as well as epithelial pro-growth factor, And other factors in the pathogenesis of meaningful.In this paper, we will make a brief review of several other aspects related to the pathogenesis of peptic ulcer.I-HSV infection and peptic ulcer Warren isolated from the vagus ganglion I-HSV. Latent I-HSV is activated by the vagus nerve can reach the digestive tract, causing herpes esophagitis and other gastrointestinal herpes lesions such lesions occur on the basis of immunocompromised, general