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根据电生理学的究研,室上性心动过速的发病机理可分为自主性异位节律点和折返激动两种。异位节律点在心房或房室交界处。折返激动通路可在房室结,窦房结或为异常房室传导通路。大多数异常房室传导通路可由体表心电图得以发现,因其可产生提示于激综合征的P-R间期缩短和QRS间期延长。予激综合征病人发作室上性心动过速时,其激动折返通路一般包括通过房室结-希氏束的前向性传导和经过异常通路的逆向性传导。最近有报导某些参与折返激动的异常传导通路不能向前传导,
According to electrophysiological studies, the pathogenesis of supraventricular tachycardia can be divided into two kinds of independent ectopic rhythm point and reentry stimulation. Ectopic rhythm points in the atrium or atrioventricular junction. Reentry excitement pathway in the atrioventricular node, sinus node or abnormal ventricular conduction path. Most of the abnormal AV conduction pathways can be found on the surface ECG because of the shortening of the P-R interval and the prolongation of the QRS interval, which are indicative of irritable bowel syndrome. In patients with preeclamptic tachycardia, the inflammatory reentry pathway generally involves anterior conduction through atrioventricular node-amygdalus bundle and retrograde conduction through an anomalous pathway. It has recently been reported that some abnormal conduction pathways involved in rejuvenation can not be conducted forwardly,