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预先反复短暂缺血可以减轻后续长期缺血所造成的组织损伤，这种现象被称为缺血预处理（PC）[1]，其细胞保护机制尚不明确。本工作在培养的乳兔心肌细胞缺氧复氧（A／R）损伤模型上观察缺氧PC的作用及蛋白激酶C（PKC）抑制剂H，和钙调素抑制剂三氟拉咦（TFP）对PC的影响，以探讨
Premature and repeated short-term ischemia can reduce tissue damage caused by subsequent long-term ischemia, a phenomenon known as ischemic preconditioning (PC) [1], whose cytoprotective mechanisms remain unclear. In this study, we observed the effect of hypoxic PC and protein kinase C (PKC) inhibitor H and calmodulin inhibitor trifloxystril (TFP) on the cultured rabbit myocardial cell model of hypoxia-reoxygenation injury (A / R) ) On the impact of PC to explore