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宿主與寄生蟲之關係基於二者之間有否適合之生理環境。宿主每因寄生蟲之侵犯,則産生特殊之抵抗,亦即所謂免疫。以免疫過程言,宿主本身則有先天舆後天免疫之區別。先天免疫之原理,現時尚多不明,蓋其各種關係之複雜,决非一單純之因素所致成。宿主之免疫多因寄生蟲種類之不同,以及傳染程度之不一,免疫現象遂有區別。寄生蟲之本質對於宿主所引起之免疫作用亦有重要之影響。Schilling氏與Schreck氏(1930)曾報告錐蟲之生物本質可能失去對血淸抗體反應作用,而産生所謂耐血淸性(Serum fast)或後發株,(relapse strain)。在治療中亦可産生耐藥物性株(drug fast strains)。如試驗室
The relationship between host and parasite is based on whether there is a suitable physiological environment between the two. Each host suffers from a special resistance due to parasite invasion, which is called immunity. According to the immune process, the host itself has the difference of congenital acquired immunity. The principle of innate immunity, the current fashion is not clear, cover the complexity of its various relationships, by no means a simple factor. The immunity of the host is mostly due to the different types of parasites and the degree of infection. There are differences in the immune phenomena. The nature of the parasite also has an important impact on the host’s immune function. Schilling’s and Schreck’s (1930) have reported that the biological nature of trypanosomes may lose their response to blood stasis antibodies, resulting in so-called serum fastness or relapse strain. Drug fast strains can also be produced during treatment. Such as laboratory