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肿瘤坏死因子α(tumornecrosisfactor-alpha,TNFα)和可溶性白细胞介素2受体(solubleinter-leukin-2receptorIL-2R)是重要的炎症和免疫反应介质。TNFα与多种免疫病理过程有关,参与脑损伤机制[1]。已证明在许多感染性疾病时,患者血清及尿中sIL-2R含量升高。但对TNFα和sIL-2R与
Tumor necrosis factor-alpha (TNFα) and solubleinter-leukin-2 receptor IL-2R are important mediators of inflammation and immune response. TNFα and a variety of immune pathological processes involved in brain injury mechanism [1]. Serum and urinary sIL-2R levels have been shown to be elevated in many infectious diseases. But for TNFα and sIL-2R and