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肾病活动期骨密度明显低于正常同龄组已为许多学者所证实，由于与白蛋白结合钙从尿中丢失及肾病综合征患儿血清25-羟维生素D3水平降低，使血清钙不同程度地下降，刺激甲状务腺亢进，而使骨质脱钙[1]，导致代谢性骨病的发生，本文以入院时所测骨密度为基点，经口服强的松治疗1个
Nephropathy in active BMD was significantly lower than the normal age group has been confirmed by many scholars, due to loss of albumin-bound calcium from the urine and children with nephrotic syndrome serum 25-hydroxy vitamin D3 levels lower serum calcium decreased to varying degrees , To stimulate thyroid gland hyperthyroidism, leaving the decalcification of bone [1], leading to the occurrence of metabolic bone disease, the paper measured at admission bone mineral density as the base, the treatment of oral prednisone 1