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关于急性黄疸性肝炎和暴发性肝功衰竭的病因研究很少,这些病因可能是甲肝(HAY)、乙肝(HBV)、非甲非乙(NANB)以及少见的巨细胞病毒(CMV)或EB病毒(EBV)。本文对一年间住院的急性黄疸性肝炎和暴发性肝功衰竭的小儿进行了病因的实验室研究。材料与方法1987年1月～12月住院观察。47例,12岁以下的黄疸小儿。具有急性肝炎的症状,全面体检后进行全血计数、肝功能检验以及血清学调查。如果以前肝功正常,突然出现肝脏病症状的患者在8周内出现肝性脑病,即可诊断暴发性肝功衰竭。分别通过抗HAV-IgM抗—CMV-IgM或抗—EBV—IgM(VCA)的检出,表明患者近期感染了HAV、CMV、EBV。在急性和恢复期之间血浆中抗—CMV-IgG的滴定度
There are few studies on the etiology of acute jaundice hepatitis and fulminant hepatic failure, which may be caused by HAY, HBV, NANB, and rare cytomegalovirus (CMV) or Epstein-Barr virus (EBV). In this paper, a year of hospitalized acute jaundice hepatitis and fulminant hepatic failure in children etiological laboratory studies. Materials and Methods January 1987 ~ December hospitalization. 47 cases, children under 12 years of jaundice. Has symptoms of acute hepatitis, full blood count, liver function tests, and serological surveys after a full physical examination. If the previous normal liver function, sudden onset of liver symptoms in patients with hepatic encephalopathy within 8 weeks, you can diagnose fulminant liver failure. Detection of anti-HAV-IgM anti-CMV-IgM or anti-EBV-IgM (VCA), respectively, indicates that patients have recently been infected with HAV, CMV, and EBV. Titers of anti-CMV-IgG in plasma between acute and convalescent phases