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動物於注射四氧嘧啶(alloxan)後,產生初期高血糖和低血糖,最後形成永久性高血糖。一般認為初期高血糖的產生和腦下垂體,腎上腺及肝臟有關,低血糖係由胰島β細胞受損傷放出已形成的胰島素所引起。中樞神經機能活動的改變可以影響内外環境的各種動因對機體引起的反應,因此,中樞神經在戊巴比妥鈉麻醉下是否也能影響四氧嘧啶引起的初期血糖变化,是值得研究的。
After alloxan injection, animals produce initial hyperglycemia and hypoglycemia, which eventually leads to permanent hyperglycemia. Generally believed that the initial high blood sugar and pituitary gland, adrenal and liver-related, hypoglycemia by the islet β-cell injury caused by the release of insulin has been formed. Changes in central nervous system activity can affect the internal and external environment caused by a variety of motivation on the body’s response, therefore, whether central nervous system anesthesia in pentobarbital can affect alloxan-induced changes in the initial blood glucose is worth studying.