烧伤休克期心脏固有舒缩功能降低是心输出量减少的原因之一。而心脏的舒缩功能对细胞外Ca~(2+)浓度有高度的依赖性。为进一步探讨心脏固有舒缩功能降低的机制,本实验选用250～300g的豚鼠,用3%凝固汽油造成35%TBAⅢ°烧伤。于伤后8小时用常钙台氏液(含2.5mmolCa~(++)/L)和高钙台氏液(含3.2mmolCa~(++)/L)灌流离体心脏。用多导记录仪 One of the reasons for the decrease of cardiac output is the decrease of cardiac intrinsic systolic and diastolic function in burn shock stage. The cardiac systolic and diastolic function of the extracellular Ca ~ (2+) concentration is highly dependent. In order to further explore the mechanism of the reduction of cardiac intrinsic systolic and diastolic function, guinea pigs of 250-300g were selected in this experiment, and 35% TBA Ⅲ ° burns were induced with 3% coagulated gasoline. Eight hours after injury, isolated hearts were perfused with normal calcium Typhoid solution (containing 2.5 mmol Ca ~ (++) / L) and high calcium Tyrode solution (containing 3.2 mmol Ca ~ (++) / L) With multi-guided recorder